GliSODin® Inhibits Cellular Oxidative Stress

MORRISTOWN, N.J.—Superoxide dismutase (as GliSODin®, www.glisodin.org) inhibits proliferation of tumor cells by lowering inflammation and limits susceptibility of cells to oxidative stress during illness, according to a new study published in the British Journal of Cancer (94, 6:854-62, 2006) and recent research from St. Petersburg State Medical Academy in Russia.

In the first study, researchers used a mouse model of inflammationdriven progression of tumor cells to measure the potential of GliSODin to inhibit active oxygen species-mediated tumor progression. The results suggested GliSODin prevented tumor progression promoted by inflammation by scavenging inflammatory cell-derived superoxide anions.

The second study showed GliSODin lowered cellular oxidative stress when added to treatment modalities in 30 patients with cardiovascular, gastrointestinal and skin diseases.

The researchers concluded GliSODin helped to promote faster response and improvement in the general somatic status of the patients compared to treatment alone.