Tea: Beyond the Brewed Cup References

May 23, 2005

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May 23, 2005 - Natural Products Industry INSIDER
"Beyond the Brewed Cup" References

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2. Sano J et al. Effects of green tea intake on the development ofcoronary heart disease. Circ J. 68, 7:665-70, 2004. http://www.jstage.jst.go.jp/browse/circj

3. Brown MD. "Green tea (Camellia sinensis) extract and its possiblerole in the prevention of cancer." Altern Med Rev. 4, 5:360-70,1999. www.thorne.com/altmedrev

4. Yang YC et al. "The protective effect of habitual tea consumption onhypertension." Arch Intern Med. 164, 14:1534-40, 2004. http://archinte.ama-assn.org

5. Tokunaga S et al. Green tea consumption and serum lipids andlipoproteins in a population of healthy workers in Japan. Ann Epidemiol. 12,3:157-65, 2002. http://www.annalsofepidemiology.org/

6. Yang TT et al. Inhibitory effect of Chinese green tea on endothelialcell-induced LDL oxidation. Atherosclerosis. 148, 1:67-73, 2000. www.sciencedirect.com/science/journal/00219150

7. Osada K et al. Tea catechins inhibit cholesterol oxidation accompanyingoxidation of low density lipoprotein in vitro. Comp Biochem Physiol CToxicol Pharmacol. 128, 2:153-64, 2001.

8. Davies MJ et al. Black tea consumption reduces total and LDLcholesterol in mildly hypercholesterolemic adults. J Nutr. 133,10:3298S-3302S, 2003. www.nutrition.org

9. Maron DJ et al. Cholesterol-lowering effect of a theaflavin-enrichedgreen tea extract: a randomized controlled trial. Arch Intern Med.163, 12:1448-53, 2003. http://archinte.ama-assn.org/

10. Erba D et al. Effectiveness of moderate green tea consumption onantioxidative status and plasma lipid profile in humans. J Nutr Biochem.16,3:144-9, 2005. http://www.journals.elsevierhealth.com/periodicals/jnb/home

11. Huxley RR and Neil HA. The relation between dietary flavonol intakeand coronary heart disease mortality: a meta-analysis of prospective cohortstudies. Eur J Clin Nutr. 57, 8:904-8, 2003. http://www.nature.com/ejcn/index.html

12. Geleijnse JM et al. Inverse association of tea and flavonoid intakeswith incident myocardial infarction: the Rotterdam Study. Am J Clin Nutr.75, 5:880-6, 2002. www.acjn.org

13. Duffy SJ et al. Short- and long-term black tea consumption reversesendothelial dysfunction in patients with coronary artery disease. Circulation.104, 2:151-6, 2001. http://circ.ahajournals.org

14. Ludwig A et al. "The tea flavonoid epigallocatechin-3-gallatereduces cytokine-induced VCAM-1 expression and monocyte adhesion to endothelialcells." Biochem Biophys Res Comm. 316, 3:659-65, 2004. www.sciencedirect.com/science/journal/0006291X

15. Chyu KY et al. "Differential effects of green tea-derived catechinon developing versus established atherosclerosis in apolipoprotein E-nullmice." Circulation. 109, 20:2448-53, 2004. http://circ.ahajournals.org/

16. Dufresne CJ, Farnworth ER. A review of latest research findings on thehealth promotion properties of tea. J Nutr Biochem.12(7):404-421,2001.

17. Nakazato T et al. Catechin, a green tea component, rapidly inducesapoptosis of myeloid leukemic cells via modulation of reactive oxygen speciesproduction in vitro and inhibits tumor growth in vivo. Haematologica.90(3):317-25, 2005.

18. Sengupta A et al. Tea can protect against aberrant crypt fociformation during azoxymethane induced rat colon carcinogenesis. J Exp ClinCancer Res. 22(2):185-91, 2003.

19. Santana-Rios G et al. Potent antimutagenic activity of white tea incomparison with green tea in the Salmonella assay. Mutat Res.495(1-2):61-74, 2001.

20. Bettuzzi S et al. The chemopreventive action of catechins in the TRAMPmouse model of prostate carcinogenesis is accompanied by clusterinover-expression. Carcinogenesis. 25, 11:2217-24, 2004. http://carcin.oupjournals.org

21. Lu YP et al. Inhibitory effects of orally administered green tea,black tea, and caffeine on skin carcinogenesis in mice previously treated withultraviolet B light (high-risk mice): relationship to decreased tissue fat. CancerRes. 61, 13:5002-9, 2001. http://cancerres.aacrjournals.org

22. Nihal M et al. Anti-proliferative and proapoptotic effects of(-)-epigallocatechin-3-gallate on human melanoma: possible implications for thechemoprevention of melanoma. Int J Cancer. 114, 4:513-21, 2005. http://www3.interscience.wiley.com/cgi-bin/jhome/29331

23. Nomura M et al. Inhibition of ultraviolet B-induced AP-1 activation bytheaflavins from black tea. Mol Carcinog. 28, 3:148-55, 2000.

24. Cao J et al. Chemopreventive effects of green and black tea onpulmonary and hepatic carcinogenesis. Fundam Appl Toxicol. 29,2:244-50, 1996.

25. Ahn WS et al. A major constituent of green tea, EGCG, inhibits thegrowth of a human cervical cancer cell line, CaSki cells, through apoptosis,G(1) arrest, and regulation of gene expression. DNA Cell Biol. 22,3:217-24, 2003.

26. Santana-Rios G et al. Inhibition by white tea of2-amino-1-methyl-6-phenylimidazo[4,5-b]pyridine-induced colonic aberrant cryptsin the F344 rat. Nutr Cancer. 41(1-2): 98-103, 2001.

27. Dashwood WM et al. Inhibition of beta-catenin/Tcfactivity by white tea, green tea, and epigallocatechin-3-gallate (EGCG): minorcontribution of H2O2 at physiological relevant EGCG concentrations. BiochemBiophys Res Commun. 296, 3: 584-8. 2002.

28. Han MK. "Epigallocatechin gallate, a constituent of green tea,suppresses cytokine-induced pancreatic beta-cell damage." Exp Mol Med.35, 2:136-9, 2003.

29. Waltner-Law ME et al. "Epigallocatechin gallate, a constituent ofgreen tea, represses hepatic glucose production." J Biol Chem. 277,38:34933-40, 2002. www.jbc.org

30. Wu LY et al. "Green tea supplementation ameliorates insulinresistance and increases glucose transporter IV content in a fructose-fed ratmodel." Eur J Nutr. 43, 2:116-24, 2004. http://link.springer.de/link/service/journals/00394/

31. Tsuneki H et al. "Effect of green tea on blood glucose levels andserum proteomic patterns in diabetic (db/db) mice and on glucose metabolism inhealthy humans." BMC Pharmacol. 4, 1:18, 2004.

32. Unno K et al. "Suppressive effect of green tea catechins onmorphologic and functional regression of the brain in aged mice with acceleratedsenescence (SAMP10)." Exp Gerontol. 39, 7:1027-34, 2004.

33. Choi YT et al. "The green tea polyphenol (-)-epigallocatechingallate attenuates beta-amyloid-induced neurotoxicity in cultured hippocampalneurons." Life Sci. 70, 5:603-14, 2001. www.sciencedirect.com/science/journal/00243205

34. Mandel S, Youdim MB. "Catechin polyphenols: neurodegeneration andneuroprotection in neurodegenerative diseases." Free Rad Biol Med.37, 3:304-17, 2004. www.elsevier.com/locate/freeradbiomed

35. Mandel S et al. "Cell signaling pathways in the neuroprotectiveactions of the green tea polyphenol (-)-epigallocatechin-3-gallate: implicationsfor neurodegenerative diseases." J Neurochem. 88, 6:1555-69, 2004. http://highwire.stanford.edu/jneurochem.shtml?uri=/

36. Wang X et al. "Green tea epigallocatechin gallate: a naturalinhibitor of fatty-acid synthase." Biochem Biophys Res Commun. 288,5:1200-6, 2001. http://www.sciencedirect.com/science/journal/0006291X

37. Murase T et al. "Beneficial effects of tea catechins on diet-inducedobesity: stimulation of lipid catabolism in the liver." Int J Obes RelatMetab Disord. 26, 11:1459-64, 2002. www-us.ebsco.com/online/OnlineTitles.asp

38. Haramizu T et al. Green tea extract improves endurance capacity andincreases muscle lipid oxidation in mice. Am J Physiol Regul Integr CompPhysiol. 288, 3:R708-15, 2005. www.ajpregu.physiology.org.

39. Nagao T et al. Ingestion of a tea rich in catechins leads to areduction in body fat and malondialdehyde-modified LDL in men. Amer J ClinNutr. 81, 1:122-29, 2005. www.ajcn.org.

40. Sugiyama T and Sadzuka Y. Theanine, a specific glutamate derivative ingreen tea, reduces the adverse reactions of doxorubicin by changing theglutathione level. Cancer Lett. 212, 2:177-84, 2004. http://www.journals.elsevierhealth.com/periodicals/can/home

41. Nagasawa k et al. Possible involvement of group I mGluRs inneuroprotective effect of theanine. Biochem Biophys Res Commun. 320,1:116-22, 2004. www.sciencedirect.com/web-editions/journal/0006291X

42. Sayama K et al. Anti-obesity effects of three major components ofgreen tea, catechins, caffeine and theanine, in mice. In Vivo. 18,1:55-62, 2004.

43. Kudo M et al. Effects of quercetin and sunphenon on responses ofcancer cells to heat shock damage. Exp Mol Pathol. 66, 1:66-75, 1999.

44. Buetler TM, et al. Green tea extract decreases muscle necrosis in mdxmice and protects against reactive oxygen species. Am J Clin Nutr. 75,4:749-53, 2002. www.ajcn.org

45. Linke AB et al. Black tea extract and dental caries formation inhamsters. Intl J Food Sci Nutr. 54, 1:89-95, 2003.

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